Gonadal steroid and chronic morphine treatment do not change the posttranslational processing of β-endorphin in the rat brain
- 1 January 1989
- journal article
- research article
- Published by Elsevier in Life Sciences
- Vol. 44 (9) , 591-601
- https://doi.org/10.1016/0024-3205(89)90191-4
Abstract
No abstract availableThis publication has 47 references indexed in Scilit:
- Desensitization of Brain Opiate Receptor Mechanisms by Gonadal Steroid Treatments that Stimulate Luteinizing Hormone Secretion*Endocrinology, 1988
- A Decline in Endogenous Opioid Influence during the Steroid-Induced Hypersecretion of Luteinizing Hormone in the Rat*Endocrinology, 1986
- Modulation of Endogenous Opioid Influence on Luteinizing Hormone Secretion by Progesterone and Estrogen*Endocrinology, 1983
- Neuropeptides derived from pro-opiocortin: behavioral, physiological, and neurochemical effects.Physiological Reviews, 1982
- Opiate binding properties of naturally occurring N- and C-Terminus modified beta-endorphinsPeptides, 1981
- Influence of N-terminal acetylation and C-terminal proteolysis on the analgesic activity of β-endorphinBiochemical Journal, 1980
- Selective conversion of β-endorphin into peptides related to γ- and α-endorphinNature, 1980
- Structure and Biosynthesis of Pro-Adrenocorticotropin/Endorphin and Related Peptides*Endocrine Reviews, 1980
- Endorphins are stored in biologically active and inactive forms: isolation of α-N-acetyl peptidesNature, 1979
- C fragment of lipotropin has a high affinity for brain opiate receptorsNature, 1976