AGONAL NATURE OF THE CEREBRAL RING HEMORRHAGES

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Abstract
CEREBRAL ring hemorrhages are seen in a variety of diseases, such as fat embolism, laceration of the brain, acute Plasmodium falciparum malaria, postvaccinial and parainfectious encephalitides, pernicious anemia, aplastic anemia and leukemia, and intoxications (barbiturates, neoarsphenamine, arsphenamine), as reviewed by Kirschbaum,1 Dietrich,2 Weimann,3 Lindau,4 Dürck,5 Wolff,6 Winblad,7 Sjövall,8 and others. These hemorrhages may occur in such large numbers throughout the white matter that the hemorrhagic tendency is considered one of the important features of the disease process. This justifies the use of the term brain purpura (Schmidt9), acute hemorrhagic leucoencephalitis (Alexander and Putnam,10 Hurst,11 Henson and Russell12), and acute necrotizing hemorrhagic encephalopathy (Adams, Cammermeyer, and Denny-Brown13). The effusions of blood are thought to take place in diseases in which the nerves to the vessels have been paralyzed and stasis has thereby been furthered, with diapedesis hemorrhages, as

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