Adrenergic Nervous System in Sodium Metabolism

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Abstract
WHEN dietary sodium is reduced to very low values the renal excretion of sodium normally decreases in three or four days to levels that approach zero. The mechanisms responsible for the renal adjustments to such a change in sodium intake are not entirely clear. Although a fall in the intake of sodium can increase the production of aldosterone, the decrease in urinary sodium may occur while aldosterone excretion (and presumably secretion) is still normal.1 Furthermore, when the intake of sodium is not restricted, large doses of aldosterone or other sodium-retaining steroids usually do not decrease urinary sodium to values as . . .