Striatal quinolinic acid lesions increase [3H]WIN 35,428 binding to the dopamine transporter
- 31 December 1992
- journal article
- Published by Elsevier in Neurochemistry International
- Vol. 21 (4) , 581-584
- https://doi.org/10.1016/0197-0186(92)90091-5
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- Nigral dopamine type-1 receptors are reduced in Huntington's disease: A postmortem autoradiographic study using [3H]SCH 23390 and correlation with [3H]forskolin bindingExperimental Neurology, 1990
- Autoradiographic localization of cocaine binding sites by [3H]CFT ([3H]WIN 35,428) in the monkey brainSynapse, 1990
- Localization of nigrostriatal dopamine receptor subtypes and adenylate cyclaseBrain Research Bulletin, 1988
- D-1 dopamine receptor changes after striatal quinolinic acid lesionEuropean Journal of Pharmacology, 1987
- Intracerebral quinolinic acid injection in the rat: Effects on dopaminergic neuronsBrain Research, 1986
- Replication of the neurochemical characteristics of Huntington's disease by quinolinic acidNature, 1986
- Quinolinic Acid: An Endogenous Metabolite That Produces Axon-Sparing Lesions in Rat BrainScience, 1983
- Compensatory bilateral changes in dopamine turnover after striatal kainate lesionNature, 1980
- Kainic acid-induced neurotoxicity in the striatum: A histofluorescent studyBrain Research, 1979
- Lesion of striatal neurons with kainic acid provides a model for Huntington's choreaNature, 1976