Physical Interaction with Human Tumor-derived p53 Mutants Inhibits p63 Activities
Open Access
- 1 May 2002
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 277 (21) , 18817-18826
- https://doi.org/10.1074/jbc.m201405200
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- A Subset of Tumor-Derived Mutant Forms of p53 Down-Regulate p63 and p73 through a Direct Interaction with the p53 Core DomainMolecular and Cellular Biology, 2001
- From p63 to p53 across p73FEBS Letters, 2001
- A gain of function p53 mutant promotes both genomic instability and cell survival in a novel p53‐null mammary epithelial cell mode.The FASEB Journal, 2000
- Activation of the insulin-like growth factor II transcription by aflatoxin B1 induced p53 mutant 249 is caused by activation of transcription complexes; implications for a gain-of-function during the formation of hepatocellular carcinomaOncogene, 2000
- High level expression of ΔN-p63: a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)?Oncogene, 2000
- A role of cyclin G in the process of apoptosisOncogene, 1999
- Mutant p53 protein expression interferes with p53-independent apoptotic pathwaysOncogene, 1998
- Lonely No More: p53 Finds Its Kin in a Tumor Suppressor HavenCell, 1997
- Induction of the growth inhibitor IGF-binding protein 3 by p53Nature, 1995
- Regulation of mdm2 expression by p53: alternative promoters produce transcripts with nonidentical translation potential.Genes & Development, 1994