Co-administration of progabide inhibits haloperidol-induced oral dyskinesias in rats
- 25 February 1992
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 212 (1) , 43-49
- https://doi.org/10.1016/0014-2999(92)90070-k
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Primate models of movement disorders of basal ganglia originTrends in Neurosciences, 1990
- Oral movements induced by interference with nigral GABA neurotransmission: Relationship to tardive dyskinesiasExperimental Neurology, 1988
- ProgabideClinical Neuropharmacology, 1985
- Association with persistent neuroleptic-induced dyskinesia of regional changes in brain GABA synthesisNature, 1984
- Oral dyskinesia in rats following brain lesions and neuroleptic drug administrationPsychopharmacology, 1982
- γ-Acetylenic GABA in Tardive DyskinesiaArchives of General Psychiatry, 1980
- Chronic blockade of dopamine receptors by antischizophrenic drugs enhances GABA binding in substantia nigraNature, 1980
- An analysis of [3H]gamma-aminobutyric acid (GABA) binding in the human brainBrain Research, 1979
- Acute and chronic haloperidol treatment: Comparison of effects on nigral dopaminergic cell activityLife Sciences, 1978
- An experimental model of tardive dyskinesiaJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1972