Regulation of c-Jun/JunB heterodimers mediated by Epstein-Barr virus encoded latent membrane protein 1 on p16

This publication has 21 references indexed in Scilit:

• Heterodimer formation between c-Jun and Jun B proteins mediated by Epstein Barr virus encoded latent membrane protein 1
  Science China Life Sciences, 2005
• AP-1 complexes containing cJun and JunB cause cellular transformation of Rat1a fibroblasts and share transcriptional targets
  Oncogene, 2003
• Evaluation of hypermethylated tumor suppressor genes as tumor markers in mouth and throat rinsing fluid, nasopharyngeal swab and peripheral blood of nasopharygeal carcinoma patient
  International Journal of Cancer, 2003
• JunB negatively regulates AP-1 activity and cell proliferation of malignant mouse keratinocytes
  Zeitschrift für Krebsforschung und Klinische Onkologie, 2001
• Jun, the oncoprotein
  Oncogene, 2001
• Regulation of the G1 phase of the mammalian cell cycle
  Cell Research, 2000
• Activation of the cJun N-terminal kinase (JNK) pathway by the Epstein-Barr virus-encoded latent membrane protein 1 (LMP1)
  Oncogene, 1998
• JunB differs from c-Jun in its DNA-binding and dimerization domains, and represses c-Jun by formation of inactive heterodimers.
  Genes & Development, 1993
• A rapid micrqpreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells
  Nucleic Acids Research, 1991
• Purification and Biochemical Characterization of the Promoter-Specific Transcription Factor, Sp1
  Science, 1986