RecQ4 Facilitates UV Light-induced DNA Damage Repair through Interaction with Nucleotide Excision Repair Factor Xeroderma Pigmentosum Group A (XPA)
Open Access
- 1 October 2008
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 283 (43) , 29037-29044
- https://doi.org/10.1074/jbc.m801928200
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Possible involvement of RecQL4 in the repair of double-strand DNA breaks in Xenopus egg extractsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 2007
- A Positive Involvement of RecQL4 in UV-Induced S-Phase ArrestDNA and Cell Biology, 2006
- The Rothmund-Thomson gene product RECQL4 localizes to the nucleolus in response to oxidative stressExperimental Cell Research, 2006
- ATR-dependent checkpoint modulates XPA nuclear import in response to UV irradiationOncogene, 2006
- The N-Terminal Noncatalytic Region of Xenopus RecQ4 Is Required for Chromatin Binding of DNA Polymerase α in the Initiation of DNA ReplicationMolecular and Cellular Biology, 2006
- RECQL4-deficient cells are hypersensitive to oxidative stress/damage: Insights for osteosarcoma prevalence and heterogeneity in Rothmund-Thomson syndromeBiochemical and Biophysical Research Communications, 2006
- Phosphorylation of Nucleotide Excision Repair Factor Xeroderma Pigmentosum Group A by Ataxia Telangiectasia Mutated and Rad3-Related–Dependent Checkpoint Pathway Promotes Cell Survival in Response to UV IrradiationCancer Research, 2006
- The human Rothmund-Thomson syndrome gene product, RECQL4, localizes to distinct nuclear foci that coincide with proteins involved in the maintenance of genome stabilityJournal of Cell Science, 2005
- Protein kinase C isoform levels in normal and sodium dodecyl sulphate-irritated mouse skinBritish Journal of Dermatology, 1998
- Rothmund-Thomson syndrome: Review of the world literatureJournal of the American Academy of Dermatology, 1992