Protein kinase C δ activates IκB-kinase α to induce the p53 tumor suppressor in response to oxidative stress
- 1 October 2007
- journal article
- Published by Elsevier in Cellular Signalling
- Vol. 19 (10) , 2088-2097
- https://doi.org/10.1016/j.cellsig.2007.06.002
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- PKCδ signaling: Mechanisms of DNA damage response and apoptosisCellular Signalling, 2007
- Enabling Death by the Abl Tyrosine Kinase: Mechanisms for Nuclear Shuttling of c-Abl in Response to DNA DamageCell Cycle, 2005
- JNK phosphorylation of 14-3-3 proteins regulates nuclear targeting of c-Abl in the apoptotic response to DNA damageNature Cell Biology, 2005
- Role of Proteolytic Activation of Protein Kinase Cδ in Oxidative Stress-Induced ApoptosisAntioxidants and Redox Signaling, 2003
- Tyrosine Phosphorylation of Protein Kinase Cδ Is Essential for Its Apoptotic Effect in Response to EtoposideMolecular and Cellular Biology, 2002
- Interaction between Protein Kinase C δ and the c-Abl Tyrosine Kinase in the Cellular Response to Oxidative StressJournal of Biological Chemistry, 2000
- Activation of protein kinase C δ by the c-Abl tyrosine kinase in response to ionizing radiationOncogene, 1998
- Regulation of DNA damage-induced apoptosis by the c-Abl tyrosine kinaseProceedings of the National Academy of Sciences, 1997
- Proteolytic Activation of Protein Kinase C δ by an ICE/CED 3-like Protease Induces Characteristics of ApoptosisThe Journal of Experimental Medicine, 1996
- The molecular heterogeneity of protein kinase C and its implications for cellular regulationNature, 1988