Acquired Isodisomy for chromosome 13 is common in AML, and associated with FLT3-itd mutations
- 20 October 2005
- journal article
- other
- Published by Springer Nature in Leukemia
- Vol. 19 (12) , 2355-2358
- https://doi.org/10.1038/sj.leu.2403988
Abstract
No abstract availableKeywords
This publication has 8 references indexed in Scilit:
- Genome-Wide Single Nucleotide Polymorphism Analysis Reveals Frequent Partial Uniparental Disomy Due to Somatic Recombination in Acute Myeloid LeukemiasCancer Research, 2005
- The role of FLT3 in haematopoietic malignanciesNature Reviews Cancer, 2003
- Analysis of FLT3 length mutations in 1003 patients with acute myeloid leukemia: correlation to cytogenetics, FAB subtype, and prognosis in the AMLCG study and usefulness as a marker for the detection of minimal residual diseaseBlood, 2002
- Analysis of FLT3-activating mutations in 979 patients with acute myelogenous leukemia: association with FAB subtypes and identification of subgroups with poor prognosisBlood, 2002
- The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the United Kingdom Medical Research Council AML 10 and 12 trialsBlood, 2001
- Development and implementation of a new rapid aneuploidy diagnostic service within the UK National Health Service and implications for the future of prenatal diagnosisThe Lancet, 2001
- Genomic structure of human FLT3: implications for mutational analysisBritish Journal of Haematology, 2001
- FLT3 internal tandem duplication mutations in adult acute myeloid leukaemia define a high-risk groupBritish Journal of Haematology, 2000