Reduction of Myocardial-Infarct Size

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Abstract
From the description of the syndrome by Herrick in 1912, until the early 1960's, the treatment of acute coronary occlusion and myocardial infarction was extremely limited. The major objectives were restricted to providing adequate physical rest to let the damaged muscle heal and to prevent myocardial rupture, preventing thromboembolic complications, and treating complications such as heart failure and arrhythmias after they had arisen. The advent of the coronary-care unit set the stage for the second era of therapy of this extremely common and hazardous condition. The aggressive and prophylactic use of newer anti-arrhythmic agents, the application of external electrical counter-shock . . .