NMDA and Non-NMDA Receptor-Mediated Excitotoxicity Are Potentiated in Cultured Striatal Neurons by Prior Chronic Depolarization
- 1 September 1999
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 159 (1) , 283-296
- https://doi.org/10.1006/exnr.1999.7135
Abstract
No abstract availableKeywords
This publication has 126 references indexed in Scilit:
- THE ENDOGENEOUS AGONIST QUINOLINIC ACID AND THE NON ENDOGENOUS HOMOQUINOLINIC ACID DISCRIMINATE BETWEEN NMDAR2 RECEPTOR SUBUNITSNeurochemistry International, 1996
- Changes in gene expression of AMPA-selective glutamate receptor subunits induced by status epilepticus in rat brainNeurochemistry International, 1994
- Cyclothiazide Treatment Unmasks AMPA Excitotoxicity in Rat Primary Hippocampal CulturesJournal of Neurochemistry, 1993
- Efflux of 45calcium from cultured primary astrocytes: Effects of glutamate receptor agonists and antagonistsNeuropharmacology, 1990
- Differential expression of excitatory amino acid receptor subtypes in cultured cerebellar neuronsNeuron, 1990
- Prolonged exposure to submicromolar concentrations of quinolinic acid causes excitotoxic damage in organotypic cultures of rat corticostriatal systemNeuroscience Letters, 1989
- The expression of γ-aminobutyric acid and leu-enkephalin immunoreactivity in primary monolayer cultures of rat striatumDevelopmental Brain Research, 1988
- Glutamate neurotoxicity in cortical cell culture is calcium dependentNeuroscience Letters, 1985
- II. Excitotoxic models for neurodegenerative disordersLife Sciences, 1984
- Temporal profile of neuronal damage in a model of transient forebrain ischemiaAnnals of Neurology, 1982