Insulin-Like Growth Factor I: The Yin and Yang of Muscle Atrophy
Open Access
- 1 November 2004
- journal article
- editorial
- Published by The Endocrine Society in Endocrinology
- Vol. 145 (11) , 4803-4805
- https://doi.org/10.1210/en.2004-1037
Abstract
No abstract availableThis publication has 28 references indexed in Scilit:
- Role of the Insulin-Like Growth Factor I Decline in the Induction of Atrogin-1/MAFbx during Fasting and DiabetesEndocrinology, 2004
- Signaling pathways weigh in on decisions to make or break skeletal muscleCurrent Opinion in Clinical Nutrition and Metabolic Care, 2004
- The IGF-1/PI3K/Akt Pathway Prevents Expression of Muscle Atrophy-Induced Ubiquitin Ligases by Inhibiting FOXO Transcription FactorsMolecular Cell, 2004
- Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle AtrophyCell, 2004
- Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivoNature Cell Biology, 2001
- CachexiaAnnals of Internal Medicine, 2000
- Regulation of Components of the Ubiquitin System by Insulin-Like Growth Factor I and Growth Hormone in Skeletal Muscle of Rats Made Catabolic with DexamethasoneEndocrinology, 1999
- Mechanisms of Muscle Wasting — The Role of the Ubiquitin–Proteasome PathwayNew England Journal of Medicine, 1996
- Amino Acid Metabolism in Human Cancer CachexiaAnnual Review of Nutrition, 1990
- Influence of glucocorticoids on skeletal muscle proteolysis in normal and diabetic-adrenalectomized eviscerated ratsMetabolism, 1990