Synergistic Activation of Caspase-3 by m-Calpain after Neonatal Hypoxia-Ischemia

This publication has 58 references indexed in Scilit:

• Caspases – controlling intracellular signals by protease zymogen activation
  Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 2000
• Procaspase-3 and Poly(ADP)ribose Polymerase (PARP) Are Calpain Substrates
  Biochemical and Biophysical Research Communications, 1999
• Cleavage of the calpain inhibitor, calpastatin, during apoptosis
  Cell Death & Differentiation, 1998
• Apoptosis in a Neonatal Rat Model of Cerebral Hypoxia-Ischemia
  Stroke, 1998
• Simultaneous Degradation of αII- and βII-Spectrin by Caspase 3 (CPP32) in Apoptotic Cells
  Journal of Biological Chemistry, 1998
• Apoptosis and necrosis in the newborn piglet brain following transient cerebral hypoxia–ischaemia
  Neuropathology and Applied Neurobiology, 1997
• Protease Involvement in Fodrin Cleavage and Phosphatidylserine Exposure in Apoptosis
  Journal of Biological Chemistry, 1996
• Fodrin degradation and subcellular distribution of calpains after neonatal rat cerebral hypoxic-ischemia
  Brain Research, 1995
• Calpain inhibitor AK295 protects neurons from focal brain ischemia. Effects of postocclusion intra-arterial administration.
  Stroke, 1994
• Evidence supporting a role for programmed cell death in focal cerebral ischemia in rats.
  Stroke, 1993