ApoA-II maintains HDL levels in part by inhibition of hepatic lipase: studies in apoA-II and hepatic lipase double knockout mice

This publication has 29 references indexed in Scilit:

• Dramatically decreased high density lipoprotein cholesterol, increased remnant clearance, and insulin hypersensitivity in apolipoprotein A-II knockout mice suggest a complex role for apolipoprotein A-II in atherosclerosis susceptibility
  Proceedings of the National Academy of Sciences, 1996
• Mild Dyslipidemia in Mice following Targeted Inactivation of the Hepatic Lipase Gene
  Published by Elsevier ,1995
• Human ApoA-II inhibits the hydrolysis of HDL triglyceride and the decrease of HDL size induced by hypertriglyceridemia and cholesteryl ester transfer protein in transgenic mice.
  Journal of Clinical Investigation, 1994
• Human HDL cholesterol levels are determined by apoA-I fractional catabolic rate, which correlates inversely with estimates of HDL particle size. Effects of gender, hepatic and lipoprotein lipases, triglyceride and insulin levels, and body fat distribution.
  Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1994
• Plasma high density lipoproteins. Metabolism and relationship to atherogenesis.
  Journal of Clinical Investigation, 1990
• High-density lipoprotein cholesterol and cardiovascular disease. Four prospective American studies.
  Circulation, 1989
• Associations of high-density lipoprotein subclasses and apolipoproteins with ischemic heart disease and coronary atherosclerosis
  American Heart Journal, 1987
• Incidence of Coronary Heart Disease and Lipoprotein Cholesterol Levels
  JAMA, 1986
• High density lipoprotein metabolism.
  Journal of Lipid Research, 1984
• High density lipoprotein as a protective factor against coronary heart disease
  The American Journal of Medicine, 1977