α-Latrotoxin, Acting via Two Ca2+-dependent Pathways, Triggers Exocytosis of Two Pools of Synaptic Vesicles
Open Access
- 1 November 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (48) , 44695-44703
- https://doi.org/10.1074/jbc.m108088200
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Latrotoxin Stimulates Secretion in Permeabilized Cells by Regulating an Intracellular Ca2+- and ATP-dependent EventPublished by Elsevier ,2000
- Tetramerisation of α-latrotoxin by divalent cations is responsible for toxin-induced non-vesicular release and contributes to the Ca2+-dependent vesicular exocytosis from synaptosomesBiochimie, 2000
- Newly Synthesized Phosphatidylinositol Phosphates Are Required for Synaptic Norepinephrine but Not Glutamate or γ-Aminobutyric Acid (GABA) ReleaseJournal of Biological Chemistry, 1998
- Microtubules and Microfilaments Participate in the Inhibition of Synaptosomal Noradrenaline Release by Tetanus ToxinJournal of Neurochemistry, 1997
- Definition of the Readily Releasable Pool of Vesicles at Hippocampal SynapsesNeuron, 1996
- Estimates for the pool size of releasable quanta at a single central synapse and for the time required to refill the pool.Proceedings of the National Academy of Sciences, 1995
- Effect of α‐Latrotoxin on Acetylcholine Release and Intracellular Ca2+ Concentration in Synaptosomes: Na+‐Dependent and Na+‐Independent ComponentsJournal of Neurochemistry, 1993
- α‐Latrotoxin Releases Both Vesicular and Cytoplasmic Glutamate from Isolated Nerve TerminalsJournal of Neurochemistry, 1990
- 4‐Aminobutyrate Can Be Released Exocytotically from Guinea‐Pig Cerebral Cortical SynaptosomesJournal of Neurochemistry, 1987
- Ca2+-dependent recycling of synaptic vesicles at the frog neuromuscular junction.The Journal of cell biology, 1980