Improved endothelial function by the thromboxane a2receptor antagonist s 18886 in patients with coronary artery disease treated with aspirin
- 1 April 2003
- journal article
- clinical trial
- Published by Elsevier in Journal of the American College of Cardiology
- Vol. 41 (7) , 1198-1204
- https://doi.org/10.1016/s0735-1097(03)00048-2
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Assessment of forearm vasodilator responses to acetylcholine and albuterol by strain gauge plethysmography: reproducibility and influence of strain gauge placementPublished by Wiley ,2001
- The Thromboxane Receptor Antagonist S18886 but Not Aspirin Inhibits Atherogenesis in Apo E–Deficient MiceArteriosclerosis, Thrombosis, and Vascular Biology, 2000
- Transcellular activation of platelets and endothelial cells by bioactive lipids in platelet microparticles.Journal of Clinical Investigation, 1997
- Systemic nature of endothelial dysfunction in atherosclerosisThe American Journal of Cardiology, 1995
- Vasodilation to acetylcholine in primary and secondary forms of human hypertension.Hypertension, 1993
- Glomerular actions of a free radical-generated novel prostaglandin, 8-epi-prostaglandin F2 alpha, in the rat. Evidence for interaction with thromboxane A2 receptors.Journal of Clinical Investigation, 1992
- Impaired endothelium-mediated vasodilation in the peripheral vasculature of patients with congestive heart failureJournal of the American College of Cardiology, 1992
- Endothelium-derived contracting factors.Hypertension, 1992
- Endothelium-dependent contractions to calcium ionophore A23187, arachidonic acid, and acetylcholine in canine basilar arteries.Stroke, 1988
- Endothelium-dependent contractions to acetylcholine in the aorta of the spontaneously hypertensive rat.Hypertension, 1986