Response of thromboxane B2, malondialdehyde and platelet sensitivity to 3 weeks low-dose aspirin (ASA) in healthy volunteers
- 1 February 1989
- journal article
- research article
- Published by Elsevier in Thrombosis Research
- Vol. 53 (3) , 261-269
- https://doi.org/10.1016/0049-3848(89)90101-1
Abstract
No abstract availableThis publication has 16 references indexed in Scilit:
- Cumulative inhibitory effect of low-dose aspirin on vascular prostacyclin and platelet thromboxane production in patients with atherosclerosis.Circulation, 1985
- Low-dose aspirin (ASA) renders human platelets more vulnerable to inhibition of aggregation by prostacyclin (PGI2)Prostaglandins, Leukotrienes and Medicine, 1983
- PHARMACOLOGY OF ANTIPLATELET DRUGS AND CLINICAL TRIALS ON THROMBOSIS PREVENTION: A DIFFICULT LINKThe Lancet, 1982
- Selective Cumulative Inhibition of Platelet Thromboxane Production by Low-dose Aspirin in Healthy SubjectsJournal of Clinical Investigation, 1982
- Aspirin inhibits platelet function independent of the acetylation of ciclo-oxygenaseThrombosis Research, 1982
- Inhibition of Prostacyclin and Platelet Thromboxane A2after Low-Dose AspirinNew England Journal of Medicine, 1981
- Low dose aspirin and inhibition of thromboxane B2 production in healthy subjectsThrombosis Research, 1980
- ASPIRIN: A PARADOXICAL EFFECT ON BLEEDING-TIMEThe Lancet, 1978
- Inhibition of platelet prostaglandin synthetase by oral aspirin.Journal of Clinical Investigation, 1978
- The effect of salicylates on the hemostatic properties of platelets in manJournal of Clinical Investigation, 1968