How important is the protein phosphorylation pathway in the toxic expression of dioxin-type chemicals?
- 19 July 1994
- journal article
- review article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 48 (2) , 215-224
- https://doi.org/10.1016/0006-2952(94)90089-2
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Comparative Ability of Various PCBs, PCDFs, and TCDD to Induce Cytochrome P450 1A1 and 1A2 Activity Following 4 Weeks of TreatmentFundamental and Applied Toxicology, 1993
- Cloning of a Factor Required for Activity of the Ah (Dioxin) ReceptorScience, 1991
- Analysis of CYP1A1 promoter function by transcription in vitroMolecular Carcinogenesis, 1991
- Genetic and Molecular Aspects of 2,3,7,8-Tetra-Chlorodibenzo-P-Dioxin ActionAnnual Review of Pharmacology and Toxicology, 1990
- Dioxins and the ah receptorAmerican Journal of Industrial Medicine, 1989
- Activation of transcription as a general mechanism of 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin actionMolecular Carcinogenesis, 1989
- Alteration of rat hepatic plasma membrane functions by 2,3,7,8-tetrachlorodibenzo-p-dioxin(TCDD)Archives of Environmental Contamination and Toxicology, 1984
- 2,3,7,8-Tetrachlorodibenzo-p-Dioxin and Related Halogenated Aromatic Hydrocarbons: Examination of the Mechanism of ToxicityAnnual Review of Pharmacology and Toxicology, 1982
- Genetic differences in the induction of monooxygenase activities by polycyclic aromatic compoundsPharmacology & Therapeutics, 1979
- Results of a two-year chronic toxicity and oncogenicity study of 2,3,7,8-tetrachlorodibenzo-p-dioxin in ratsToxicology and Applied Pharmacology, 1978