Destabilization of the RB Tumor Suppressor Protein and Stabilization of p53 Contribute to HPV Type 16 E7-Induced Apoptosis

This publication has 50 references indexed in Scilit:

• The retinoblastoma gene product protects E2F-1 from degradation by the ubiquitin-proteasome pathway.
  Genes & Development, 1996
• Degradation of E2F by the ubiquitin-proteasome pathway: regulation by retinoblastoma family proteins and adenovirus transforming proteins.
  Genes & Development, 1996
• Deficiency of retinoblastoma protein leads to inappropriate S-phase entry, activation of E2F-responsive genes, and apoptosis.
  Proceedings of the National Academy of Sciences, 1995
• A Mutational Analysis of the Amino Terminal Domain of the Human Papillomavirus Type 16 E7 Oncoprotein
  Virology, 1994
• Apoptosis or retinoblastoma: alternative fates of photoreceptors expressing the HPV-16 E7 gene in the presence or absence of p53.
  Genes & Development, 1994
• Elevated Wild-Type p53 Protein Levels in Human Epithelial Cell Lines Immortalized by the Human Papillomavirus Type 16 E7 Gene
  Virology, 1994
• Requirement for a functional Rb-1 gene in murine development
  Nature, 1992
• Functional analysis of human papillomavirus type 16 E7 by complementation with adenovirus E1A mutants
  Journal of General Virology, 1992
• Induction of apoptosis in fibroblasts by c-myc protein
  Cell, 1992
• Negative charge at the casein kinase II phosphorylation site is important for transformation but not for Rb protein binding by the E7 protein of human papillomavirus type 16.
  Proceedings of the National Academy of Sciences, 1991