A Spontaneous Point Mutation Produces Monoamine Oxidase A/B Knock-out Mice with Greatly Elevated Monoamines and Anxiety-like Behavior
Open Access
- 1 September 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (38) , 39645-39652
- https://doi.org/10.1074/jbc.m405550200
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- Dual Functions of Transcription Factors, Transforming Growth Factor-β-inducible Early Gene (TIEG)2 and Sp3, Are Mediated by CACCC Element and Sp1 Sites of Human Monoamine Oxidase (MAO) B GeneJournal of Biological Chemistry, 2004
- Abnormal anxiety‐related behavior in serotonin transporter null mutant mice: the influence of genetic backgroundGenes, Brain and Behavior, 2003
- Decreased Methylation and Transcription Repressor Sp3 Up-regulated Human Monoamine Oxidase (MAO) B Expression during Caco-2 DifferentiationJournal of Biological Chemistry, 2003
- Influence of Life Stress on Depression: Moderation by a Polymorphism in the 5-HTT GeneScience, 2003
- Anxiety and increased aggression as pacemakers of depressionActa Psychiatrica Scandinavica, 1998
- Effects on Anxiety and Memory of Systemic and Intra-Amygdala Injection of 5-HT3 Receptor Antagonist BRL 46470ANeuropsychobiology, 1996
- Enhanced Aggressive Behavior in Mice Lacking 5-HT 1B ReceptorScience, 1994
- Anxiogenic-like effects induced by stimulation of dopamine receptorsPharmacology Biochemistry and Behavior, 1993
- Clinical, biochemical, and neuropsychiatric evaluation of a patient with a contiguous gene syndrome due to a microdeletion Xp11.3 including the Norrie disease locus and monoamine oxidase (MAOA and MAOB) genesAmerican Journal of Medical Genetics, 1992
- Hydroxylation of phenylethylamine by rat liver preparations. Inhibition studiesGeneral Pharmacology: The Vascular System, 1989