Y-box Protein-1 Is the Crucial Mediator of Antifibrotic Interferon-γ Effects
Open Access
- 1 January 2006
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 281 (3) , 1784-1795
- https://doi.org/10.1074/jbc.m510215200
Abstract
No abstract availableKeywords
This publication has 54 references indexed in Scilit:
- Cell Type-Specific Intervention of Transforming Growth Factor β/Smad Signaling Suppresses Collagen Gene Expression and Hepatic Fibrosis in MiceGastroenterology, 2005
- Interferon-γ Interferes with Transforming Growth Factor-β Signaling through Direct Interaction of YB-1 with Smad3Journal of Biological Chemistry, 2003
- The pleiotropic functions of the Y‐box‐binding protein, YB‐1BioEssays, 2003
- Mechanisms of TGF-β Signaling from Cell Membrane to the NucleusCell, 2003
- Nuclear localization of Y-box factor YB1 requires wild-type p53Oncogene, 2003
- The Y-box Binding Protein YB-1 Suppresses Collagen α1(I) Gene Transcription via an Evolutionarily Conserved Regulatory Element in the Proximal PromoterJournal of Biological Chemistry, 2001
- The role of TGFβ1 in initiating hepatic stellate cell activation in vivoPublished by Elsevier ,1999
- A Synergistic Interaction of Transcription Factors AP2 and YB-1 Regulates Gelatinase A Enhancer-dependent TranscriptionPublished by Elsevier ,1998
- Induction of Inhibitory Smad6 and Smad7 mRNA by TGF-β Family MembersBiochemical and Biophysical Research Communications, 1998
- IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses.Journal of Clinical Investigation, 1998