Hypoxanthine-Guanine Phosphoribosyltransferase Mutant Glioma Cells: Diminished Monoamine Oxidase Activity

Abstract
A defective capability of cultured rat glioma cells to reutilize purine bases (hypoxanthine-guanine phosphoribosyltransferase deficiency) was associated with a reduced capacity to oxidatively deaminate serotonin and tryptamine. The mutant glioma cells were also more sensitive to the cytotoxic effects of serotonin than were normal cells.