Intercellular communication may facilitate apoptosis: Implications for tumor promotion
- 1 September 1994
- journal article
- review article
- Published by Wiley in Molecular Carcinogenesis
- Vol. 11 (1) , 8-12
- https://doi.org/10.1002/mc.2940110103
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- Phorbol esters inhibit apoptosis in IL-2-dependent T lymphocytesPublished by Elsevier ,2004
- Endogenous and exogenous modulation of gap junctional intercellular communication: Toxicological and pharmacological implicationsLife Sciences, 1993
- p53 is required for radiation-induced apoptosis in mouse thymocytesNature, 1993
- Studies of the relationship between cell proliferation and cell death. II. Early gene expression during concanavalin A-induced proliferation or dexamethasone-induced apoptosis of rat thymocytesBiochemical and Biophysical Research Communications, 1992
- Apoptosis and the regulation of cell numbers in normal and neoplastic tissues: an overviewCancer and Metastasis Reviews, 1992
- Incorporation of the gene for a cell-cell channel protein into transformed cells leads to normalization of growthThe Journal of Membrane Biology, 1991
- Inhibition of radiation-induced apoptosis in vitro by tumor promotersBiochemical and Biophysical Research Communications, 1988
- Inhibition of intercellular communication between mouse hepatocytes by tumor promoters*1, *2Toxicology and Applied Pharmacology, 1987
- Modulation of cell communication and carcinogenesis.The Japanese Journal of Physiology, 1985
- Phenotypic diversity in experimental hepatomas: the concept of partially blocked ontogeny. The 10th Walter Hubert LectureBritish Journal of Cancer, 1978