Glutathione deficit during development induces anomalies in the rat anterior cingulate GABAergic neurons: Relevance to schizophrenia
- 1 June 2006
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 22 (3) , 624-637
- https://doi.org/10.1016/j.nbd.2006.01.003
Abstract
No abstract availableKeywords
This publication has 101 references indexed in Scilit:
- Schizophrenia genes, gene expression, and neuropathology: on the matter of their convergenceMolecular Psychiatry, 2004
- Mitochondrial dysfunction in schizophrenia: evidence for compromised brain metabolism and oxidative stressMolecular Psychiatry, 2004
- GABAergic neuronal subtypes in the human frontal cortex — development and deficits in schizophreniaJournal of Chemical Neuroanatomy, 2001
- Schizophrenia: the fundamental questionsBrain Research Reviews, 2000
- Modulation of neuronal and recombinant GABAA receptors by redox reagentsThe Journal of Physiology, 1999
- γ‐Glutamylglutamine and Taurine Concentrations Are Decreased in the Cerebrospinal Fluid of Drug‐Naive Patients with Schizophrenic DisordersJournal of Neurochemistry, 1995
- Transient co-localization of calretinin, parvalbumin, and calbindin-D28k in developing visual cortex of monkeyJournal of Neurocytology, 1995
- Three distinct subpopulations of GABAergic neurons in rat frontal agranular cortexBrain Research, 1994
- NMDA receptor channels: Subunit-specific potentiation by reducing agentsNeuron, 1994
- Ultrastructural analysis of somatostatin‐immunoreactive neurons and synapses in the temporal and occipital cortex of the macaque monkeyJournal of Comparative Neurology, 1989