β-Arrestin Mediates β1-Adrenergic Receptor-Epidermal Growth Factor Receptor Interaction and Downstream Signaling
Open Access
- 1 July 2009
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 284 (30) , 20375-20386
- https://doi.org/10.1074/jbc.m109.005793
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- How Does Arrestin Assemble MAPKs into a Signaling Complex?Journal of Biological Chemistry, 2009
- Vasopressin up-regulates the expression of growth-related immediate-early genes via two distinct EGF receptor transactivation pathwaysCellular Signalling, 2008
- β-Blockers alprenolol and carvedilol stimulate β-arrestin-mediated EGFR transactivationProceedings of the National Academy of Sciences, 2008
- Clathrin-Mediated Internalization Is Essential for Sustained EGFR Signaling but Dispensable for DegradationPublished by Elsevier ,2008
- β-Arrestin Scaffolding of Phosphatidylinositol 4-Phosphate 5-Kinase Iα Promotes Agonist-stimulated Sequestration of the β2-Adrenergic ReceptorJournal of Biological Chemistry, 2008
- Persistent transactivation of EGFR and ErbB2/HER2 by protease-activated receptor-1 promotes breast carcinoma cell invasionOncogene, 2008
- Epidermal Growth Factor Receptor and Protein Kinase C Signaling to ERK2Published by Elsevier ,2008
- Ubiquitination of β-Arrestin Links Seven-transmembrane Receptor Endocytosis and ERK ActivationJournal of Biological Chemistry, 2007
- Mitogenic signaling pathways induced by G protein‐coupled receptorsJournal of Cellular Physiology, 2007
- Functional specialization of β-arrestin interactions revealed by proteomic analysisProceedings of the National Academy of Sciences, 2007