Therapy with antibody against leukocyte integrin VLA-4 (CD49d) is effective and safe in virus-facilitated experimental allergic encephalomyelitis
- 31 January 1997
- journal article
- Published by Elsevier in Journal of Neuroimmunology
- Vol. 72 (1) , 95-105
- https://doi.org/10.1016/s0165-5728(96)00158-0
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- The pathophysiologic role of alpha 4 integrins in vivo.Journal of Clinical Investigation, 1994
- Monocyte rolling, arrest and spreading on IL-4-activated vascular endothelium under flow is mediated via sequential action of L-selectin, beta 1-integrins, and beta 2-integrins.The Journal of cell biology, 1994
- Animal modelsAnnals of Neurology, 1994
- Traffic signals for lymphocyte recirculation and leukocyte emigration: The multistep paradigmCell, 1994
- Adhesion molecule expression on cerebrospinal fluid T lymphocytes: Evidence for common recruitment mechanisms in multiple sclerosis, aseptic meningitis, and normal controlsAnnals of Neurology, 1993
- Inhibition of experimental autoimmune encephalomyelitis by an antibody to the intercellular adhesion molecule ICAM‐1Annals of Neurology, 1993
- ICAM-1-dependent pathway is not critically involved in the inflammatory process of autoimmune encephalomyelitis or in cytokine-induced inflammation of the central nervous systemJournal of Neuroimmunology, 1993
- Surface expression of alpha 4 integrin by CD4 T cells is required for their entry into brain parenchyma.The Journal of Experimental Medicine, 1993
- Transfer of diabetes in mice prevented by blockade of adhesion-promoting receptor on macrophagesNature, 1990
- Cellular immune reactivity within the CNSTrends in Neurosciences, 1986