Alterations in Cerebral Blood Flow and Glucose Utilization in Mice Overexpressing the Amyloid Precursor Protein
- 1 February 2002
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 9 (1) , 61-68
- https://doi.org/10.1006/nbdi.2001.0460
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- SOD1 rescues cerebral endothelial dysfunction in mice overexpressing amyloid precursor proteinNature Neuroscience, 1999
- Genetic Modification of the Phenotypes Produced by Amyloid Precursor Protein Overexpression in Transgenic MiceHuman Molecular Genetics, 1997
- Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic MiceScience, 1996
- Age-related CNS disorder and early death in transgenic FVB/N mice overexpressing Alzheimer amyloid precursor proteinsNeuron, 1995
- Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor proteinNature, 1995
- Early-onset Alzheimer's disease caused by mutations at codon 717 of the β-amyloid precursor protein geneNature, 1991
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991
- Altered angioarchitecture in selected areas of brains with Alzheimer's diseaseActa Neuropathologica, 1990
- Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid proteinBiochemical and Biophysical Research Communications, 1984