Increasing the number of tandem estrogen response elements increases the estrogenic activity of a tamoxifen analogue

This publication has 25 references indexed in Scilit:

• The biological action of cDNAs from mutated estrogen receptors transfected into breast cancer cells
  Cancer Letters, 1995
• Agonist-antagonist activity of anti-estrogens in the human breast cancer cell line MCF-7: an hypothesis for the interaction with a site distinct from the estrogen binding site
  Molecular and Cellular Endocrinology, 1994
• Investigation of the Mechanism of Tamoxifen-Stimulated Breast Tumor Growth With Nonisomerizable Analogues of Tamoxifen and Metabolites
  JNCI Journal of the National Cancer Institute, 1993
• Functional analysis of the human estrogen receptor using a phenotypic transactivation assay in yeast
  Gene, 1992
• A far upstream estrogen response element of the ovalbumin gene contains several half-palindromic 5′-TGACC-3′ motifs acting synergistically
  Cell, 1992
• Systemic treatment of early breast cancer by hormonal, cytotoxic, or immune therapy
  The Lancet, 1992
• Cis- and trans-acting elements of the estrogen-regulated vitellogenin gene B1 of Xenopus laevis
  Journal of Steroid Biochemistry, 1989
• Tamoxifen withdrawal response. Report of a case
  Archives of internal medicine (1960), 1989
• Contrasting ability of antiestrogens to inhibit MCF-7 growth stimulated by estradiol or epidermal growth factor
  European Journal of Cancer and Clinical Oncology, 1989
• CLINICAL SIGNIFICANCE OF TAMOXIFEN WITHDRAWAL RESPONSE
  The Lancet, 1987