Evidence for a genetic aetiology in reading disability of twins
- 1 October 1987
- journal article
- Published by Springer Nature in Nature
- Vol. 329 (6139) , 537-539
- https://doi.org/10.1038/329537a0
Abstract
Reading disability (dyslexia) is a major social, educational, and mental health problem. Although estimates of prevalence vary, up to 10-15% of school-age children have severe reading deficits in spite of average intelligence and adequate educational opportunity. That reading disability may have a constitutional basis has long been recognized, and results of twin and family studies suggest that one or more of its forms may be heritable; however, definitive evidence for a genetic aetiology has not been reported. Establishing a heritable basis for reading disability could suggest possible causes, give improved risk estimates, facilitate early diagnosis, and provide validity tests for ostensible subtypes. In this report, we apply a recently developed multiple regression analysis to data collected from a sample of 64 pairs of identical twins and 55 pairs of fraternal twins, in which at least one member of the pairs is reading disabled, and present evidence for a significant genetic aetiology.Keywords
This publication has 4 references indexed in Scilit:
- Multiple regression analysis of twin data obtained from selected samplesGenetic Epidemiology, 1986
- Multiple regression analysis of twin dataBehavior Genetics, 1985
- THE IDENTIFICATION AND PREVALENCE OF SPECIFIC READING RETARDATIONBritish Journal of Educational Psychology, 1983
- Parent-offspring resemblance for cognitive abilities in the Colorado Adoption Project: Biological, adoptive, and control parents and one-year-old childrenIntelligence, 1981