Abstract
The selec-tive binding of S35 by the healing fractured bones of rats was used to determine the catabolic effect of dihydrotachysterol (AT-10) and the anabolic action of 17-ethyl-19-nortestosterone (Nilevar). In the dosages used, AT-10 lowered the uptake of S35 by fractured bone, expressed as the ratio of the radioactivity of fractured (F) and intact (I) humeri 3 weeks after fracture, from 2.03 in control animals to 0.87. When Nilevar was administered in an attempt to curb the effect of AT-10, the F/I ratio was 1.65. It is concluded that AT-10 blocks and 17-ethyl-19-nortestosterone stimulates the synthesis of mucopolysaccharides in newly formed collagen tissue. 17-ethyl-19-nortestosterone also protects this tissue against catabolic effect of AT-10.

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