Ref‐1/Ape is critical for formation of the hypoxia‐inducible transcriptional complex on the hypoxic response element of the rat pulmonary artery endothelial cell VEGF gene
Open Access
- 14 April 2004
- journal article
- fj express-summary
- Published by Wiley in The FASEB Journal
- Vol. 18 (9) , 986-988
- https://doi.org/10.1096/fj.03-1160fje
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Redox factor-1 contributes to the regulation of progression from G0/G1 to S by PDGF in vascular smooth muscle cellsAmerican Journal of Physiology-Heart and Circulatory Physiology, 2003
- Regulation of ornithine decarboxylase and polyamine import by hypoxia in pulmonary artery endothelial cellsAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 2002
- Hypoxia promotes oxidative base modifications in the pulmonary artery endothelial cell VEGF geneThe FASEB Journal, 2001
- Going APE over ref-1Mutation Research/DNA Repair, 2000
- Free radical production in hypoxic pulmonary artery smooth muscle cellsAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 2000
- Redox-Regulated Recruitment of the Transcriptional Coactivators CREB-Binding Protein and SRC-1 to Hypoxia-Inducible Factor 1αMolecular and Cellular Biology, 2000
- Regulation of Transcription by Hypoxia Requires a Multiprotein Complex That Includes Hypoxia-Inducible Factor 1, an Adjacent Transcription Factor, and p300/CREB Binding ProteinMolecular and Cellular Biology, 1998
- Activation of Hypoxia-inducible Transcription Factor Depends Primarily upon Redox-sensitive Stabilization of Its α SubunitJournal of Biological Chemistry, 1996
- An essential role for p300/CBP in the cellular response to hypoxiaProceedings of the National Academy of Sciences, 1996
- Transcriptional Regulation of the Rat Vascular Endothelial Growth Factor Gene by HypoxiaJournal of Biological Chemistry, 1995