Reperfusion Injury as the Mechanism of Brain Damage after Perinatal Asphyxia
- 1 May 1997
- journal article
- review article
- Published by Springer Nature in Pediatric Research
- Vol. 41 (5) , 599-606
- https://doi.org/10.1203/00006450-199705000-00001
Abstract
Pediatric Research publishes original papers, invited reviews, and commentaries on the etiologies of diseases of children and disorders of development, extending from molecular biology to epidemiology. Use of model organisms and in vitro techniques relevant to developmental biology and medicine are acceptable, as are translational human studies.Keywords
This publication has 122 references indexed in Scilit:
- Microglia activation after neonatal hypoxic-ischemiaDevelopmental Brain Research, 1995
- Purification and immunohistochemical tissue localization of human xanthine oxidaseBiochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 1993
- Glutamate triggers internucleosomal DNA cleavage in neuronal cellsBiochemical and Biophysical Research Communications, 1991
- Cerebral blood flow, cross-brain oxygen extraction, and fontanelle pressure after hypoxic-ischemic injury in newborn infantsThe Journal of Pediatrics, 1991
- Vasoparalysis associated with brain damage in asphyxiated term infantsThe Journal of Pediatrics, 1990
- Tissue Destruction by NeutrophilsNew England Journal of Medicine, 1989
- Xanthine oxidase produces hydrogen peroxide which contributes to reperfusion injury of ischemic, isolated, perfused rat hearts.Journal of Clinical Investigation, 1988
- Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog.Circulation, 1983
- Temporal profile of neuronal damage in a model of transient forebrain ischemiaAnnals of Neurology, 1982
- Uric acid provides an antioxidant defense in humans against oxidant- and radical-caused aging and cancer: a hypothesis.Proceedings of the National Academy of Sciences, 1981