β‐Cyclodextrin interacts with the Alzheimer amyloid β‐A4 peptide

Abstract
Electrospray ionisation mass spectrometry has been used to show that the synthetic 40 amino acid β-amyloid peptide (β1-40) interacts with the cyclic oligosaccharide β-cyclodextrin. This interaction, presumably with the hydrophobic aromatic moieties on the peptide, has been shown to diminish substantially the neurotoxic effects of β1-40 in a cell line.