A Novel C-Terminal Domain of Drosophila PERIOD Inhibits dCLOCK:CYCLE-Mediated Transcription
- 1 April 2003
- journal article
- research article
- Published by Elsevier in Current Biology
- Vol. 13 (9) , 758-762
- https://doi.org/10.1016/s0960-9822(03)00286-0
Abstract
No abstract availableKeywords
Funding Information
- Howard Hughes Medical Institute
- National Institutes of Health (NS 39303)
This publication has 16 references indexed in Scilit:
- Sequential Nuclear Accumulation of the Clock Proteins Period and Timeless in the Pacemaker Neurons ofDrosophila melanogasterJournal of Neuroscience, 2002
- Molecular Components of the Circadian System in DrosophilaAnnual Review of Physiology, 2001
- A TIMELESS-Independent Function for PERIOD Proteins in the Drosophila ClockNeuron, 2000
- Transport Between the Cell Nucleus and the CytoplasmAnnual Review of Cell and Developmental Biology, 1999
- PER and TIM Inhibit the DNA Binding Activity of a Drosophila CLOCK-CYC/dBMAL1 Heterodimer without Disrupting Formation of the Heterodimer: a Basis for Circadian TranscriptionMolecular and Cellular Biology, 1999
- Closing the Circadian Loop: CLOCK-Induced Transcription of Its Own Inhibitors per and timScience, 1998
- Regulation of Nuclear Entry of the Drosophila Clock Proteins Period and TimelessNeuron, 1996
- Isolation of timeless by PER Protein Interaction: Defective Interaction Between timeless Protein and Long-Period Mutant PER LScience, 1995
- Temporally regulated nuclear entry of the Drosophila period protein contributes to the circadian clockNeuron, 1995
- PAS is a dimerization domain common to Drosophila Period and several transcription factorsNature, 1993