Sustained Nitric Oxide Exposure Decreases Soluble Guanylate Cyclase mRNA and Enzyme Activity in Pulmonary Artery Smooth Muscle

This publication has 16 references indexed in Scilit:

• Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K channel by cGMP-dependent protein kinase.
  Proceedings of the National Academy of Sciences, 1994
• The nitric oxide and cGMP signal transduction system: regulation and mechanism of action
  Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1993
• Endothelial control of the pulmonary circulation in normal and chronically hypoxic rats.
  The Journal of Physiology, 1993
• Cytokines and Lipopolysaccharide Induce Nitric Oxide Synthase in Cultured Rat Pulmonary Artery Smooth Muscle
  American Journal of Respiratory Cell and Molecular Biology, 1992
• Effect of nitric oxide synthase inhibitors on hypotension in patients with septic shock
  The Lancet, 1991
• Hepatocytes produce nitrogen oxides from L-arginine in response to inflammatory products of Kupffer cells.
  The Journal of Experimental Medicine, 1989
• Role of cGMP in relaxation of vascular and other smooth muscle
  Canadian Journal of Physiology and Pharmacology, 1989
• Macrophage oxidation of L-arginine to nitrite and nitrate: nitric oxide is an intermediate
  Biochemistry, 1988
• Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide.
  Proceedings of the National Academy of Sciences, 1987
• Nitric oxide activates guanylate cyclase and increases guanosine 3′:5′-cyclic monophosphate levels in various tissue preparations
  Proceedings of the National Academy of Sciences, 1977