Eotaxin and CCR3 are up-regulated in exacerbations of chronic bronchitis

This publication has 27 references indexed in Scilit:

• INTERLEUKIN 5 RELEASE INTO ASTHMATIC AIRWAYS 4 AND 24HOURS AFTER ENDOBRONCHIAL ALLERGEN CHALLENGE: ITS RELATIONSHIP WITH EOSINOPHIL RECRUITMENT
  Cytokine, 1999
• The C-C chemokine receptor CCR3 participates in stimulation of eosinophil arrest on inflammatory endothelium in shear flow.
  Journal of Clinical Investigation, 1998
• Enhanced expression of eotaxin and CCR3 mRNA and protein in atopic asthma. Association with airway hyperresponsiveness and predominant co‐localization of eotaxin mRNA to bronchial epithelial and endothelial cells
  European Journal of Immunology, 1997
• Short-term Treatment with a Low Dose of Inhaled Fluticasone Propionate Decreases the Number of CD1a+ Dendritic Cells in Asthmatic Airways
  Pulmonary Pharmacology & Therapeutics, 1997
• Airway eosinophilia and expression of interleukin‐5 protein in asthma and in exacerbations of chronic bronchitis
  Clinical and Experimental Allergy, 1996
• Molecular cloning and characterization of a human eotaxin receptor expressed selectively on eosinophils.
  The Journal of Experimental Medicine, 1996
• Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils.
  Journal of Clinical Investigation, 1996
• Eotaxin: a potent eosinophil chemoattractant cytokine detected in a guinea pig model of allergic airways inflammation.
  The Journal of Experimental Medicine, 1994
• Inflammatory Markers in Bronchoalveolar Lavage and in Bronchial Biopsy in Asthma during Remission
  Chest, 1990