Blockade of TGF-β action ameliorates renal dysfunction and histologic progression in anti-GBM nephritis
- 1 July 2003
- journal article
- Published by Elsevier in Kidney International
- Vol. 64 (1) , 92-101
- https://doi.org/10.1046/j.1523-1755.2003.00045.x
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Blockade of TGF-β by in vivo gene transfer of a soluble TGF-β type II receptor in the muscle inhibits corneal opacification, edema and angiogenesisGene Therapy, 2000
- A Soluble Transforming Growth Factor beta Receptor Expressed in Muscle Prevents Liver Fibrogenesis and Dysfunction in RatsHuman Gene Therapy, 2000
- Glomerular epithelial–myofibroblast transdifferentiation in the evolution of glomerular crescent formationNephrology Dialysis Transplantation, 1999
- Gene therapy by transforming growth factor-β receptor-IgG Fc chimera suppressed extracellular matrix accumulation in experimental glomerulonephritisKidney International, 1999
- Inhibition of TGF-β1 expression by antisense oligonucleotides suppressed extracellular matrix accumulation in experimental glomerulonephritisKidney International, 1996
- TGF-beta induced transdifferentiation of mammary epithelial cells to mesenchymal cells: involvement of type I receptors.The Journal of cell biology, 1994
- Increased collagen synthesis and decreased collagen degradation in right ventricular hypertrophy induced by pressure overloadCardiovascular Research, 1994
- Cardiac angiotensin converting enzyme and myocardial fibrosis in the ratCardiovascular Research, 1994
- Sustained expression of TGF-β1 underlies development of progressive kidney fibrosisKidney International, 1994
- Suppression of experimental glomerulonephritis by antiserum against transforming growth factor β1Nature, 1990