Repeated physiologic stresses provide persistent cardioprotection against ischemia-reperfusion injury in rats

This publication has 18 references indexed in Scilit:

• The involvement of cytokines in the second window of ischaemic preconditioning
  British Journal of Pharmacology, 2000
• Involvement of Cytokines in the Mechanism of Whole-Body Hyperthermia-Induced Cardioprotection
  Circulation, 2000
• Monophosphoryl lipid A provides biphasic cardioprotection against ischaemia‐reperfusion injury in rat hearts
  British Journal of Pharmacology, 1999
• Exercise Provides Direct Biphasic Cardioprotection via Manganese Superoxide Dismutase Activation
  The Journal of Experimental Medicine, 1999
• Whole-Body Hyperthermia Provides Biphasic Cardioprotection Against Ischemia/Reperfusion Injury in the Rat
  Circulation, 1998
• Prolonging the Delayed Phase of Myocardial Protection: Repetitive Adenosine A1Receptor Activation Maintains Rabbit Myocardium in a Preconditioned State 11Dr. Dana is supported by a Junior Research Fellowship, and Dr. Baxter by an Intermediate Fellowship, from the British Heart Foundation, London. Continuing support (Drs. Dana and Baxter) is provided by the Hatter Foundation, London.
  Journal of the American College of Cardiology, 1998
• Myocardial protection after monophosphoryl lipid A: studies of delayed anti-ischaemic properties in rabbit heart
  British Journal of Pharmacology, 1996
• Heat-shock protein induction in rat hearts. A direct correlation between the amount of heat-shock protein induced and the degree of myocardial protection.
  Circulation, 1994
• Cardiac stress protein elevation 24 hours after brief ischemia or heat stress is associated with resistance to myocardial infarction.
  Circulation, 1993
• Delayed effects of sublethal ischemia on the acquisition of tolerance to ischemia.
  Circulation Research, 1993