Glutathione depletion and cytotoxicity by naphthalene 1,2-oxide in isolated hepatocytes
- 1 January 1989
- journal article
- research article
- Published by Elsevier in Chemico-Biological Interactions
- Vol. 71 (2-3) , 147-165
- https://doi.org/10.1016/0009-2797(89)90031-8
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Platelets and the puzzles of pulmonary pyrrolizidine poisoningToxicology and Applied Pharmacology, 1988
- Effects of chemical and enzymic probes on microsomal covalent binding of bromobenzene and derivatives. Evidence for quinones as reactive metabolitesXenobiotica, 1988
- Redox cycling and sulphydryl arylation; Their relative importance in the mechanism of quinone cytotoxicity to isolated hepatocytesChemico-Biological Interactions, 1988
- Evidence that 1-naphthol is not an obligate intermediate in the covalent binding and the pulmonary bronchiolar necrosis by naphthaleneBiochemical and Biophysical Research Communications, 1985
- Evidence that covalent binding of metabolically activated phenol to microsomal proteins is caused by oxidised products of hydroquinone and catecholChemico-Biological Interactions, 1985
- Differences in naphthalene-induced toxicity in the mouse and ratChemico-Biological Interactions, 1985
- Detection and half-life of bromobenzene-3,4-oxide in bloodXenobiotica, 1984
- Evidence for cytochrome P-450 mediated metabolism in the bronchiolar damage by naphthaleneChemico-Biological Interactions, 1982
- Bromobenzene and p-bromophenol toxicity and covalent bindingLife Sciences, 1982
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976