A γ34.5 mutant of herpes simplex 1 causes severe inflammation in the brain
- 1 January 1998
- journal article
- Published by Elsevier in Neuroscience
- Vol. 83 (4) , 1225-1237
- https://doi.org/10.1016/s0306-4522(97)00513-7
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Immune responses to adenovirus vectors in the nervous systemTrends in Neurosciences, 1996
- Inflammatory cytokines in the brain: Does the CNS shape immune responses?Immunology Today, 1994
- The herpes simplex virus type 1 strain 17+ 34.5 deletion mutant 1716 is avirulent in SCID miceJournal of General Virology, 1994
- Nervous tissue as an immune compartment: the dialect of the immune response in the CNSImmunology Today, 1994
- Characterization of the herpes simplex virus type 1 strain 17+ neurovirulence gene RL1 and its expression in a bacterial systemJournal of General Virology, 1994
- Roles of alphabeta and gammadelta T Cell Subsets in Viral ImmunityAnnual Review of Immunology, 1992
- The RL neurovirulence locus in herpes simplex virus type 2 strain HG52 plays no role in latencyJournal of General Virology, 1991
- The Herpes Simplex Virus Type 2 (HG52) Variant JH2604 Has a 1488 bp Deletion which Eliminates Neurovirulence in MiceJournal of General Virology, 1989
- A Variant of Herpes Simplex Virus Type 2 Strain HG52 with a 1.5 kb Deletion in RL between 0 to 0.02 and 0.81 to 0.83 Map Units Is Non-neurovirulent for MiceJournal of General Virology, 1989
- Genetics of natural resistance to herpesvirus infections in miceNature, 1975