Deletion of the ABL SH3 domain reactivates de-oligomerized BCR-ABL for growth factor independence

This publication has 21 references indexed in Scilit:

• Signalling through SH2 and SH3 domains
  Published by Elsevier ,2002
• Alternative signals to RAS for hematopoietic transformation by the BCR-ABL oncogene
  Cell, 1995
• Genetic requirement for Ras in the transformation of fibroblasts and hematopoietic cells by the Bcr-Abl oncogene.
  The Journal of Experimental Medicine, 1995
• Membrane targeting of the nucleotide exchange factor Sos is sufficient for activating the Ras signaling pathway
  Cell, 1994
• Differential Complementation of Bcr-Abl Point Mutants with c-Myc
  Science, 1994
• The COOH terminus of the c-Abl tyrosine kinase contains distinct F- and G-actin binding domains with bundling activity [published erratum appears in J Cell Biol 1994 Mar;124(5):865]
  The Journal of cell biology, 1994
• SH3 domains direct cellular localization of signaling molecules
  Cell, 1993
• SH2 and SH3 domains: From structure to function
  Cell, 1992
• Dominant negative MYC blocks transformation by ABL oncogenes
  Cell, 1992
• The mouse type IV c-abl gene product is a nuclear protein, and activation of transforming ability is associated with cytoplasmic localization
  Cell, 1989