Weight-of-evidence versus strength-of-evidence in toxicologic hazard identification: Di(2-ethylhexyl)phthalate (DEHP)
- 6 March 2001
- journal article
- research article
- Published by Elsevier in Toxicology
- Vol. 160 (1-3) , 219-226
- https://doi.org/10.1016/s0300-483x(00)00451-0
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Chronic Toxicity of Di(2-ethylhexyl)phthalate in RatsToxicological Sciences, 2000
- Central role of PPARα in the mechanism of action of hepatocarcinogenic peroxisome proliferatorsMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 2000
- A Cancer Risk Assessment of Di(2-ethylhexyl)phthalate: Application of the New U.S. EPA Risk Assessment GuidelinesRegulatory Toxicology and Pharmacology, 1999
- Oral Toxicity of Bis(2-Ethylhexyl) Phthalate During Pregnancy and Suckling in the Long–Evans RatFood and Chemical Toxicology, 1998
- Do Peroxisome Proliferating Compounds Pose a Hepatocarcinogenic Hazard to Humans?Regulatory Toxicology and Pharmacology, 1998
- Mechanistically-based Human Hazard Assessment of Peroxisome Proliferator-induced HepatocarcinogenesisHuman & Experimental Toxicology, 1994
- Positive regulation of the peroxisomal β‐oxidation pathway by fatty acids through activation of peroxisome proliferator‐activated receptors (PPAR)Biology of the Cell, 1993
- Control of the peroxisomal β-oxidation pathway by a novel family of nuclear hormone receptorsPublished by Elsevier ,1992
- The Potential Role of Chemically Induced Hyperplasia in the Carcinogenic Activity of the Hypolipidemic CarcinogensToxicology and Industrial Health, 1987
- Influence of fenofibrate on cellular and subcellular liver structure in hyperlipidemic patientsAtherosclerosis, 1983