Potential risk of β-blockade withdrawal in congestive heart failure due to abrupt autonomic changes
- 28 February 1999
- journal article
- clinical trial
- Published by Elsevier in International Journal of Cardiology
- Vol. 68 (2) , 171-177
- https://doi.org/10.1016/s0167-5273(98)00356-8
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Increased Cardiac Adrenergic Drive Precedes Generalized Sympathetic Activation in Human Heart FailureCirculation, 1997
- Adverse consequences of high sympathetic nervous activity in the failing human heartJournal of the American College of Cardiology, 1995
- Can power spectral analysis of heart rate variability identify a high risk subgroup of congestive heart failure patients with excessive sympathetic activation? A pilot study before and after heart transplantation.Heart, 1994
- Cardiac sympathetic nervous activity in congestive heart failure. Evidence for increased neuronal norepinephrine release and preserved neuronal uptake.Circulation, 1993
- Parasympathetic withdrawal is an integral component of autonomic imbalance in congestive heart failure: Demonstration in human subjects and verification in a paced canine model of ventricular failureJournal of the American College of Cardiology, 1991
- Hormones regulating cardiovascular function in patients with severe congestive heart failure and their relation to mortality. CONSENSUS Trial Study Group.Circulation, 1990
- Clinical and hemodynamic correlates of sympathetic nerve activity in normal humans and patients with heart failure: Evidence from direct micronenrographic recordingsJournal of the American College of Cardiology, 1990
- Assessment of autonomic regulation in chronic congestive heart failure by heart rate spectral analysisThe American Journal of Cardiology, 1988
- Direct evidence from intraneural recordings for increased central sympathetic outflow in patients with heart failure.Circulation, 1986
- Plasma Norepinephrine as a Guide to Prognosis in Patients with Chronic Congestive Heart FailureNew England Journal of Medicine, 1984