NMDA Receptor-Dependent Activation of the Small GTPase Rab5 Drives the Removal of Synaptic AMPA Receptors during Hippocampal LTD
Open Access
- 1 January 2005
- Vol. 45 (1) , 81-94
- https://doi.org/10.1016/j.neuron.2004.12.023
Abstract
No abstract availableKeywords
Funding Information
- Alzheimer's Association
- National Alliance for Research on Schizophrenia and Depression
- National Institute of Mental Health (F31-MH070205, MH070417)
- National Institute on Aging (P50-AG08671)
This publication has 77 references indexed in Scilit:
- Removal of AMPA Receptors (AMPARs) from Synapses Is Preceded by Transient Endocytosis of Extrasynaptic AMPARsJournal of Neuroscience, 2004
- Tyrosine phosphorylation of GluR2 is required for insulin-stimulated AMPA receptor endocytosis and LTDThe EMBO Journal, 2004
- Dynamics and Regulation of Clathrin Coats at Specialized Endocytic Zones of Dendrites and SpinesNeuron, 2002
- Role of ampa receptor endocytosis in synaptic plasticityNature Reviews Neuroscience, 2001
- Regulation of AMPA receptor endocytosis by a signaling mechanism shared with LTDNature Neuroscience, 2000
- Dual MAP kinase pathways mediate opposing forms of long-term plasticity at CA3–CA1 synapsesNature Neuroscience, 2000
- Dynamin-dependent endocytosis of ionotropic glutamate receptorsProceedings of the National Academy of Sciences, 1999
- Rapid redistribution of glutamate receptors contributes to long-term depression in hippocampal culturesNature Neuroscience, 1999
- A synaptic model of memory: long-term potentiation in the hippocampusNature, 1993
- The small GTPase rab5 functions as a regulatory factor in the early endocytic pathwayCell, 1992