A Common C→T Polymorphism at nt 46 in the Promoter Region of Coagulation Factor XII is Associated With Decreased Factor XII Activity
- 1 February 2001
- journal article
- Published by Elsevier in Thrombosis Research
- Vol. 101 (4) , 255-260
- https://doi.org/10.1016/s0049-3848(00)00404-7
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Fibrinolytic properties of activated FXIIEuropean Journal of Biochemistry, 1999
- Factor XII Tenri, a Novel Cross-Reacting Material Negative Factor XII Deficiency, Occurs Through a Proteasome-Mediated DegradationBlood, 1999
- Factor XIIa Is a Kinetically Favorable Plasminogen ActivatorThrombosis and Haemostasis, 1999
- A Common Genetic Polymorphism (46 C to T Substitution) in the 5′-Untranslated Region of the Coagulation Factor XII Gene Is Associated With Low Translation Efficiency and Decrease in Plasma Factor XII LevelBlood, 1998
- Hemostatic Effects of Oral Contraceptives in Women who Developed Deep-vein Thrombosis while Using Oral ContraceptivesThrombosis and Haemostasis, 1998
- Risk of Coronary Heart Disease and Activation of Factor XII in Middle-Aged MenArteriosclerosis, Thrombosis, and Vascular Biology, 1997
- Molecular basis of estrogen regulation of Hageman factor XII gene expressionEndocrinology, 1995
- Hemostatic abnormalities in acute myocardial infarction as detected by specific blood markersThrombosis Research, 1994
- John Hageman's factor and deep‐vein thrombosis: Leiden Thrombophilia StudyBritish Journal of Haematology, 1994