At the scene of ischemic brain injury: Is PARP a perp?
- 1 October 1997
- journal article
- Published by Springer Nature in Nature Medicine
- Vol. 3 (10) , 1073-1074
- https://doi.org/10.1038/nm1097-1073
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- Ischemic Brain Injury is Mediated by the Activation of Poly(ADP-Ribose)PolymeraseJournal of Cerebral Blood Flow & Metabolism, 1997
- Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemiaNature Medicine, 1997
- Genetic determinants of susceptibility to excitotoxic cell death: Implications for gene targeting approachesProceedings of the National Academy of Sciences, 1997
- Gene-targeting studies of mammalian behavior: is it the mutation or the background genotype?Trends in Neurosciences, 1996
- Additive neuroprotective effects of dextrorphan and cycloheximide in rats subjected to transient focal cerebral ischemiaBrain Research, 1996
- Mice lacking ADPRT and poly(ADP-ribosyl)ation develop normally but are susceptible to skin disease.Genes & Development, 1995
- Nitric Oxide Activation of Poly(ADP-Ribose) Synthetase in NeurotoxicityScience, 1994
- Excitotoxic cell deathJournal of Neurobiology, 1992
- Nitric oxide mediates glutamate neurotoxicity in primary cortical cultures.Proceedings of the National Academy of Sciences, 1991
- Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide.Proceedings of the National Academy of Sciences, 1990