Reversal of Proximal Tubular Dysfunction by Indomethacin

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Abstract
This paper reports a patient with salt-losing nephropathy, phosphaturia, glucosuπa, hypercalcmria and bicarbonaturia, i.e. urinary abnormalities due to proximal tubular defects. Oral indomethacin caused a complete normalization of urinary alterations. These results show that indomethacin is effective in the treatment of proximal tubular defects and suggest that this therapeutic effect is due to an overall increase in proximal tubular reabsorption.

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