IL-6 plays an obligatory role in late preconditioning via JAK?STAT signaling and upregulation of iNOS and COX-2
Open Access
- 1 October 2004
- journal article
- research article
- Published by Oxford University Press (OUP) in Cardiovascular Research
- Vol. 64 (1) , 61-71
- https://doi.org/10.1016/j.cardiores.2004.05.011
Abstract
Objective: We sought to determine whether interleukin (IL)-6 modulates myocardial infarction or the late phase of preconditioning (PC). Methods: Wild-Keywords
This publication has 31 references indexed in Scilit:
- Preconditioning the Myocardium: From Cellular Physiology to Clinical CardiologyPhysiological Reviews, 2003
- Role of interleukin‐6 for left ventricular remodeling and survival after experimental myocardial infarctionThe FASEB Journal, 2003
- Mechanism of cyclooxygenase-2 upregulation in late preconditioningJournal of Molecular and Cellular Cardiology, 2003
- Induction of Interleukin-6 Synthesis in the MyocardiumCirculation, 1995
- Hypoxic Stress Induces Cardiac Myocyte–Derived Interleukin-6Circulation, 1995
- A nitric oxide synthase inhibitor reduces inflammation, down-regulates inflammatory cytokines and enhances interleukin-10 production in carrageenin-induced oedema in mice.1994
- Induction of Nitric Oxide Synthase in Subsets of Murine Pulmonary Fibroblasts: Effect on Fibroblast Interleukin-6 ProductionClinical Immunology and Immunopathology, 1994
- Delayed effects of sublethal ischemia on the acquisition of tolerance to ischemia.Circulation Research, 1993
- Promoter of the mouse gene encoding calcium-independent nitric oxide synthase confers inducibility by interferon gamma and bacterial lipopolysaccharide.The Journal of Experimental Medicine, 1993
- The biology of interleukin-6.1992