Contribution of Estrogen Receptor α to Oncogenic K-Ras-mediated NIH3T3 Cell Transformation and Its Implication for Escape from Senescence by Modulating the p53 Pathway

This publication has 23 references indexed in Scilit:

• Opposing Effects of Ras on p53: Transcriptional Activation of mdm2 and Induction of p19ARF
  Published by Elsevier ,2000
• The Estrogen Receptor Enhances AP-1 Activity by Two Distinct Mechanisms with Different Requirements for Receptor Transactivation Functions
  Molecular Endocrinology, 1999
• The Estrogen Receptor Enhances AP-1 Activity by Two Distinct Mechanisms with Different Requirements for Receptor Transactivation Functions
  Molecular Endocrinology, 1999
• Contribution of enhanced transcriptional activation by ER to [12Val] K-Ras mediated NIH3T3 cell transformation
  Oncogene, 1997
• Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a
  Cell, 1997
• Activation of the Estrogen Receptor Through Phosphorylation by Mitogen-Activated Protein Kinase
  Science, 1995
• A biomarker that identifies senescent human cells in culture and in aging skin in vivo.
  Proceedings of the National Academy of Sciences, 1995
• Peptide growth factors elicit estrogen receptor-dependent transcriptional activation of an estrogen-responsive element
  Molecular Endocrinology, 1993
• The estrogen receptor binds tightly to its responsive element as a ligand-induced homodimer
  Cell, 1988
• Construction and applications of a highly transmissible murine retrovirus shuttle vector
  Cell, 1984